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RNA helicases remodel the spliceosome to enable pre-mRNA splicing, but their binding and mechanism of action remain poorly understood. To define helicase-RNA contacts in specific spliceosomal states, we develop purified spliceosome iCLIP (psiCLIP), which reveals dynamic helicase-RNA contacts during splicing catalysis. The helicase Prp16 binds along the entire available single-stranded RNA region between the branchpoint and 3'-splice site, while Prp22 binds diffusely downstream of the branchpoint before exon ligation, but then switches to more narrow binding in the downstream exon after exon ligation, arguing against a mechanism of processive translocation. Depletion of the exon-ligation factor Prp18 destabilizes Prp22 binding to the pre-mRNA, suggesting that proofreading by Prp22 may sense the stability of the spliceosome during exon ligation. Thus, psiCLIP complements structural studies by providing key insights into the binding and proofreading activity of spliceosomal RNA helicases.

Original publication

DOI

10.1038/s41467-021-21745-9

Type

Journal article

Journal

Nature communications

Publication Date

03/2021

Volume

12

Addresses

MRC Laboratory of Molecular Biology, Cambridge, UK.

Keywords

Spliceosomes, RNA Helicases, Ribonucleoproteins, Small Nuclear, Ribonucleoprotein, U5 Small Nuclear, Saccharomyces cerevisiae Proteins, Recombinant Proteins, RNA Precursors, RNA, Fungal, Autoantigens, Cryoelectron Microscopy, RNA Splicing, Exons, Models, Molecular, Adenosine Triphosphatases, DEAD-box RNA Helicases, RNA Splicing Factors